Deck 8: The Vitamins

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Question
The active form of vitamin A for vision is:

A) opsin.
B) retinoic acid.
C) retinal.
D) rhodopsin.
E) retinol-binding protein.
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Question
Which of the following is a major cause of childhood blindness worldwide?

A) vitamin C deficiency
B) riboflavin deficiency
C) pantothenic acid deficiency
D) vitamin A deficiency
E) rickets disease
Question
Children with measles or measles-related infections may benefit from supplementation with:

A) vitamin C.
B) vitamin A.
C) iron.
D) folate.
E) vitamin E.
Question
Vitamin A plays a role in cell differentiation. What does this mean?

A) It helps cells differentiate between the vitamins.
B) It allows cells to mature so they can perform their intended functions.
C) It promotes the storage of fat-soluble vitamins.
D) It aids in differentiating between fat- and water-soluble vitamins.
E) It causes abnormal fetal development and birth defects.
Question
Beriberi is also called the thiamin-deficiency disease.
Question
Overconsumption of the fat-soluble vitamins results in:

A) accumulation, which aids in the prevention of colds and infections.
B) no ill effects because they are excreted.
C) possible toxicity due to an accumulation in the body.
D) weight gain due to increased fat intake.
E) deficiency of water-soluble vitamins.
Question
Many of the symptoms of vitamin A deficiency, such as blindness and an increase in respiratory infections, are related to the role of vitamin A in:

A) blood clotting.
B) synthesis of visual pigments.
C) controlling osteoclast and osteoblast activity.
D) synthesis of hormones.
E) maintaining the epithelial cells.
Question
It is not absolutely necessary to consume the fat-soluble vitamins every day because they:

A) would just be excreted from the body.
B) are only absorbed in limited amounts.
C) can be stored in the body for future use.
D) would reach toxic amounts in the body.
E) cannot be absorbed every day.
Question
Vitamin A deficiency results in rickets disease in children.
Question
How are vitamins similar to carbohydrates, fats, and proteins?

A) typical roles in the body
B) organic nature
C) chemical structure
D) amounts required in the diet
E) rate of absorption
Question
Which form of vitamin A is involved in gene expression?

A) retinoic acid
B) retinol
C) retinal
D) retinol-binding protein
E) opsin
Question
In which of the following is vitamin A least likely to be involved?

A) maintaining healthy epithelial tissues
B) maintaining healthy eye tissues
C) fighting infection
D) releasing energy from the energy-yielding nutrients
E) sperm development
Question
Beta-carotene prevents which of the following from damaging cells?

A) free radicals
B) antioxidants
C) blood clots
D) vitamin E
E) calciferol
Question
Which of the following statements about fat-soluble vitamins would you be least likely to tell your client?

A) They are readily excreted from the body when consumed in excess.
B) They must be bound to proteins to travel in the blood.
C) They may be consumed less frequently than the water-soluble vitamins.
D) They may accumulate to toxic levels in the body.
E) Toxicity is more likely to occur from supplements rather than food.
Question
Excessive vitamin A during pregnancy poses a teratogenic risk. This means that:

A) it can increase the risk of birth defects.
B) it can build up in the blood and cause calcification of soft tissues.
C) it can result in a low-birth weight infant.
D) it can increase the risk of infectious disease.
E) it can result in osteomalacia.
Question
A dietary deficiency of vitamin A can produce:

A) rickets.
B) night blindness.
C) a prolonged blood-clotting time.
D) sensitivity to light.
E) birth defects.
Question
Which of the following would not be considered a fat-soluble vitamin?

A) vitamin A
B) vitamin C
C) vitamin D
D) vitamin E
E) vitamin K
Question
Bioavailability refers to the rate and extent to which a nutrient is absorbed and used.
Question
The rate and extent to which a vitamin is absorbed and used by the body is called:

A) vitamin efficiency.
B) bioavailability.
C) fortification.
D) vitamin equivalency.
E) enzymatic destruction.
Question
The fat-soluble vitamins:

A) are less stable than the water-soluble vitamins.
B) are first absorbed into the lymph.
C) are usually constituents of coenzymes.
D) are not essential.
E) are needed in frequent doses.
Question
The chemical name for vitamin E is:

A) beta-carotene.
B) cholecalciferol.
C) ascorbic acid.
D) tocopherol.
E) prothrombin.
Question
Vitamin A toxicity is likely to result from:

A) consuming too many dark green and deep orange vegetables.
B) overconsumption of whole grains.
C) consuming large amounts of fortified foods or vitamin A supplements.
D) drinking too much orange juice.
E) prescription acne treatments.
Question
Identify the food group that is the best source of beta-carotene.

A) protein foods group
B) milk group
C) vegetable group
D) grain group
E) fats and oils group
Question
Among fruits and vegetables, the best sources of beta-carotene are:

A) green or yellow, like lettuce and corn.
B) dark green or deep orange, like broccoli and sweet potatoes.
C) green, like lettuce, peas, and snap beans.
D) brightly colored, like tomatoes and lemons.
E) white, like rice and potatoes.
Question
What can be concluded from the results of research on vitamin E supplementation?

A) There is not enough evidence to recommend taking supplements to prevent heart disease.
B) Sufficient evidence exists to recommend that everyone over 40 should take a supplement.
C) Large doses are more beneficial than low doses.
D) All people who have had a heart attack should take a supplement.
E) Vitamin E can be used in place of statins in people with high cholesterol.
Question
From the list below, choose the most reliable food source of vitamin D.

A) whole grains
B) fortified breakfast cereal
C) yogurt
D) orange juice
E) raw skim milk
Question
A deficiency of which of the following vitamins can cause rickets?

A) A
B) B12
C) C
D) D
E) E
Question
The vitamin D-deficiency disease of children is:

A) xerophthalmia.
B) osteomalacia.
C) osteoporosis.
D) rickets.
E) skin cancer.
Question
Excessive intakes of vitamin D by adults may result in:

A) greater bone density.
B) mineral deposits in soft tissues such as the kidney.
C) deformity of the leg bones, ribs, and skull.
D) increased bone calcification.
E) increased risk of bone fractures.
Question
Vitamin D can be made in the body with the help of:

A) bacteria.
B) tryptophan.
C) vitamin A.
D) sunlight.
E) exercise.
Question
The effects of a deficiency of vitamin D are most readily observed in the:

A) nervous system.
B) cardiovascular system.
C) muscular system.
D) epithelial tissue.
E) skeletal system.
Question
Vitamin E:

A) functions as an effective anti-sterility agent in human beings.
B) protects vitamin A and polyunsaturated fatty acids from oxidation.
C) is needed by human beings as a dietary supplement because of the variety of functions it has in the body.
D) slows down the aging process.
E) activates blood clotting factors.
Question
Vitamin D deficiency is most likely among:

A) dark-skinned people who live in the south.
B) fair-skinned people who live in the south.
C) dark-skinned people with limited exposure to sunlight.
D) fair-skinned people with limited exposure to sunlight.
E) fair-skinned people who work outside most of the time.
Question
The main function of vitamin D in bone growth is to:

A) synthesize 7-dehydrocholesterol.
B) assist in the absorption of calcium and phosphorus.
C) mobilize calcium from the bone.
D) secrete calcitonin.
E) regulate the fat cells of adipose tissue.
Question
The major role of vitamin E in the body seems to be to:

A) aid in normal blood clotting.
B) act as an antioxidant.
C) aid in formation of normal epithelial tissue.
D) aid in protein metabolism.
E) prevent skin cancer.
Question
Which of the following factors is least likely to limit vitamin D synthesis in the body?

A) geographic location
B) season of the year
C) sunscreens
D) use of tanning beds
E) smog
Question
An excess of which of the following vitamins can cause excess calcium withdrawal from the bones?

A) A
B) B6
C) B12
D) C
E) D
Question
Osteomalacia is mainly due to a deficiency of which of the following?

A) vitamin A
B) vitamin D
C) parathyroid hormone
D) calcitonin
E) Vitamin E
Question
After 30 minutes of sun exposure, which of the following people would have synthesized more vitamin D?

A) a light-skinned person
B) a dark-skinned person
C) neither, because skin pigmentation is not related to vitamin D synthesis
D) they would both synthesize the same amount
E) a heavily-wrinkled person
Question
If your eyes have difficulty adjusting to dim light, eating which of the following foods daily might improve your vision?

A) steamed carrots
B) a peanut butter sandwich
C) brewer's yeast
D) pork chops
E) tofu
Question
Vitamin E deficiencies in human beings are associated with:

A) diseases of the pancreas.
B) low-carbohydrate weight-loss diets.
C) use of vitamin K supplements.
D) strict vegetarian (vegan) diets.
E) high-fat diets.
Question
Abuse of alcohol may lead to a severe form of thiamin deficiency called:

A) beriberi.
B) Wernicke-Korsakoff syndrome.
C) pellagra.
D) rickets.
E) atrophic gastritis.
Question
Which vitamin is synthesized in the intestine by bacteria?

A) vitamin A
B) vitamin C
C) vitamin D
D) vitamin E
E) vitamin K
Question
The most well-known role of vitamin B6 is in relation to:

A) carbohydrate and lactose metabolism.
B) fat metabolism.
C) protein and amino acid metabolism.
D) mineral metabolism.
E) cell division.
Question
Riboflavin is used by the body to:

A) facilitate energy metabolism.
B) prevent the disease pellagra.
C) provide building material for red blood cells.
D) maintain epithelial tissue.
E) synthesize niacin.
Question
Vitamin B6:

A) does not play a role in the metabolism of energy-yielding nutrients.
B) helps to regulate blood glucose.
C) is absorbed like the fat-soluble vitamins.
D) is only found in animal foods.
E) plays a major role in the formation of collagen.
Question
Thiamin is involved in:

A) formation of red blood cells.
B) blood coagulation.
C) collagen formation.
D) energy metabolism.
E) cell division.
Question
Absorption of which of the following requires the presence of intrinsic factor?

A) vitamin B6
B) vitamin D
C) vitamin B12
D) vitamin A
E) vitamin K
Question
Which vitamin can cause a pharmacological e ffect known to dilate the capillaries ?

A) thiamin
B) vitamin E
C) niacin
D) vitamin C
E) vitamin K
Question
Vitamin K has long been known for:

A) its functions in energy metabolism.
B) promoting healthy epithelial tissue.
C) its role in blood clotting.
D) enhancing calcium utilization.
E) maintaining the integrity of epithelial tissues.
Question
Which vitamin is most vulnerable to interactions with alcohol and other drugs?

A) vitamin C
B) vitamin E
C) folate
D) biotin
E) cobalamin
Question
Overconsumption of the water-soluble vitamins will likely result in:

A) a reserve supply, which will aid in the prevention of colds and infections.
B) no apparent change, because they are stored in the adipose tissues.
C) no apparent change, because they are excreted.
D) toxic accumulation in the liver.
E) a deficiency, because excess vitamins will be excreted.
Question
Which of the following statements is true of niacin?

A) It can be synthesized in the body from the amino acid tryptophan.
B) It can be used successfully to cure schizophrenia.
C) It is water soluble and therefore safe to administer in large doses.
D) It is an antioxidant.
E) A deficiency during pregnancy can lead to neural tube defects.
Question
The replacement of red blood cells and digestive tract cells depends most heavily on:

A) thiamin.
B) riboflavin.
C) niacin.
D) cobalamin.
E) folate.
Question
Which of the following groups of people is most at risk for a primary deficiency of vitamin B12?

A) the elderly
B) infants
C) athletes
D) vegans
E) pregnant women
Question
Ascorbic acid is another name for:

A) niacin.
B) thiamin.
C) vitamin C.
D) vitamin B6.
E) vitamin E.
Question
An important role of vitamin B12 is to assist folate in:

A) cell division.
B) conversion of tryptophan to niacin.
C) release of energy from food.
D) regulation of the body's use of calcium and phosphorus.
E) initiating blood clotting.
Question
Which of the following foods would make the greatest contribution to a person's intake of riboflavin?

A) oatmeal
B) milk
C) oranges
D) yellow squash
E) apples
Question
Vitamin E deficiency causes:

A) xerophthalmia.
B) rickets.
C) leukocytosis.
D) osteomalacia.
E) erythrocyte hemolysis.
Question
In which of the following foods would you find the least amount of Vitamin E?

A) margarine
B) salad dressing
C) wheat germ oil
D) butter
E) shortening
Question
Match between columns
pernicious anemia
a compound that protects other compounds from oxygen by itself reacting with oxygen.
pernicious anemia
rupture of the red blood cells, caused by vitamin E deficiency.
pernicious anemia
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
pernicious anemia
the vitamin K-deficiency disease in which blood fails to clot.
pernicious anemia
a bone disease characterized by softening of the bones.
pernicious anemia
literally, porous bones; reduced density of the bones.
pernicious anemia
the vitamin D-deficiency disease in children.
pernicious anemia
the thiamin-deficiency disease.
pernicious anemia
a metabolic disease in which crystals of uric acid precipitate in the joints.
pernicious anemia
malformations of the brain, spinal cord, or both during embryonic development.
pernicious anemia
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
pernicious anemia
the niacin-deficiency disease.
pernicious anemia
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
pernicious anemia
the vitamin C-deficiency disease.
osteoporosis
a compound that protects other compounds from oxygen by itself reacting with oxygen.
osteoporosis
rupture of the red blood cells, caused by vitamin E deficiency.
osteoporosis
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
osteoporosis
the vitamin K-deficiency disease in which blood fails to clot.
osteoporosis
a bone disease characterized by softening of the bones.
osteoporosis
literally, porous bones; reduced density of the bones.
osteoporosis
the vitamin D-deficiency disease in children.
osteoporosis
the thiamin-deficiency disease.
osteoporosis
a metabolic disease in which crystals of uric acid precipitate in the joints.
osteoporosis
malformations of the brain, spinal cord, or both during embryonic development.
osteoporosis
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
osteoporosis
the niacin-deficiency disease.
osteoporosis
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
osteoporosis
the vitamin C-deficiency disease.
pellagra
a compound that protects other compounds from oxygen by itself reacting with oxygen.
pellagra
rupture of the red blood cells, caused by vitamin E deficiency.
pellagra
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
pellagra
the vitamin K-deficiency disease in which blood fails to clot.
pellagra
a bone disease characterized by softening of the bones.
pellagra
literally, porous bones; reduced density of the bones.
pellagra
the vitamin D-deficiency disease in children.
pellagra
the thiamin-deficiency disease.
pellagra
a metabolic disease in which crystals of uric acid precipitate in the joints.
pellagra
malformations of the brain, spinal cord, or both during embryonic development.
pellagra
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
pellagra
the niacin-deficiency disease.
pellagra
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
pellagra
the vitamin C-deficiency disease.
scurvy
a compound that protects other compounds from oxygen by itself reacting with oxygen.
scurvy
rupture of the red blood cells, caused by vitamin E deficiency.
scurvy
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
scurvy
the vitamin K-deficiency disease in which blood fails to clot.
scurvy
a bone disease characterized by softening of the bones.
scurvy
literally, porous bones; reduced density of the bones.
scurvy
the vitamin D-deficiency disease in children.
scurvy
the thiamin-deficiency disease.
scurvy
a metabolic disease in which crystals of uric acid precipitate in the joints.
scurvy
malformations of the brain, spinal cord, or both during embryonic development.
scurvy
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
scurvy
the niacin-deficiency disease.
scurvy
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
scurvy
the vitamin C-deficiency disease.
niacin equivalents
a compound that protects other compounds from oxygen by itself reacting with oxygen.
niacin equivalents
rupture of the red blood cells, caused by vitamin E deficiency.
niacin equivalents
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
niacin equivalents
the vitamin K-deficiency disease in which blood fails to clot.
niacin equivalents
a bone disease characterized by softening of the bones.
niacin equivalents
literally, porous bones; reduced density of the bones.
niacin equivalents
the vitamin D-deficiency disease in children.
niacin equivalents
the thiamin-deficiency disease.
niacin equivalents
a metabolic disease in which crystals of uric acid precipitate in the joints.
niacin equivalents
malformations of the brain, spinal cord, or both during embryonic development.
niacin equivalents
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
niacin equivalents
the niacin-deficiency disease.
niacin equivalents
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
niacin equivalents
the vitamin C-deficiency disease.
osteomalacia
a compound that protects other compounds from oxygen by itself reacting with oxygen.
osteomalacia
rupture of the red blood cells, caused by vitamin E deficiency.
osteomalacia
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
osteomalacia
the vitamin K-deficiency disease in which blood fails to clot.
osteomalacia
a bone disease characterized by softening of the bones.
osteomalacia
literally, porous bones; reduced density of the bones.
osteomalacia
the vitamin D-deficiency disease in children.
osteomalacia
the thiamin-deficiency disease.
osteomalacia
a metabolic disease in which crystals of uric acid precipitate in the joints.
osteomalacia
malformations of the brain, spinal cord, or both during embryonic development.
osteomalacia
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
osteomalacia
the niacin-deficiency disease.
osteomalacia
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
osteomalacia
the vitamin C-deficiency disease.
antioxidant
a compound that protects other compounds from oxygen by itself reacting with oxygen.
antioxidant
rupture of the red blood cells, caused by vitamin E deficiency.
antioxidant
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
antioxidant
the vitamin K-deficiency disease in which blood fails to clot.
antioxidant
a bone disease characterized by softening of the bones.
antioxidant
literally, porous bones; reduced density of the bones.
antioxidant
the vitamin D-deficiency disease in children.
antioxidant
the thiamin-deficiency disease.
antioxidant
a metabolic disease in which crystals of uric acid precipitate in the joints.
antioxidant
malformations of the brain, spinal cord, or both during embryonic development.
antioxidant
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
antioxidant
the niacin-deficiency disease.
antioxidant
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
antioxidant
the vitamin C-deficiency disease.
neural tube defects
a compound that protects other compounds from oxygen by itself reacting with oxygen.
neural tube defects
rupture of the red blood cells, caused by vitamin E deficiency.
neural tube defects
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
neural tube defects
the vitamin K-deficiency disease in which blood fails to clot.
neural tube defects
a bone disease characterized by softening of the bones.
neural tube defects
literally, porous bones; reduced density of the bones.
neural tube defects
the vitamin D-deficiency disease in children.
neural tube defects
the thiamin-deficiency disease.
neural tube defects
a metabolic disease in which crystals of uric acid precipitate in the joints.
neural tube defects
malformations of the brain, spinal cord, or both during embryonic development.
neural tube defects
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
neural tube defects
the niacin-deficiency disease.
neural tube defects
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
neural tube defects
the vitamin C-deficiency disease.
free radicals
a compound that protects other compounds from oxygen by itself reacting with oxygen.
free radicals
rupture of the red blood cells, caused by vitamin E deficiency.
free radicals
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
free radicals
the vitamin K-deficiency disease in which blood fails to clot.
free radicals
a bone disease characterized by softening of the bones.
free radicals
literally, porous bones; reduced density of the bones.
free radicals
the vitamin D-deficiency disease in children.
free radicals
the thiamin-deficiency disease.
free radicals
a metabolic disease in which crystals of uric acid precipitate in the joints.
free radicals
malformations of the brain, spinal cord, or both during embryonic development.
free radicals
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
free radicals
the niacin-deficiency disease.
free radicals
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
free radicals
the vitamin C-deficiency disease.
rickets
a compound that protects other compounds from oxygen by itself reacting with oxygen.
rickets
rupture of the red blood cells, caused by vitamin E deficiency.
rickets
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
rickets
the vitamin K-deficiency disease in which blood fails to clot.
rickets
a bone disease characterized by softening of the bones.
rickets
literally, porous bones; reduced density of the bones.
rickets
the vitamin D-deficiency disease in children.
rickets
the thiamin-deficiency disease.
rickets
a metabolic disease in which crystals of uric acid precipitate in the joints.
rickets
malformations of the brain, spinal cord, or both during embryonic development.
rickets
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
rickets
the niacin-deficiency disease.
rickets
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
rickets
the vitamin C-deficiency disease.
gout
a compound that protects other compounds from oxygen by itself reacting with oxygen.
gout
rupture of the red blood cells, caused by vitamin E deficiency.
gout
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
gout
the vitamin K-deficiency disease in which blood fails to clot.
gout
a bone disease characterized by softening of the bones.
gout
literally, porous bones; reduced density of the bones.
gout
the vitamin D-deficiency disease in children.
gout
the thiamin-deficiency disease.
gout
a metabolic disease in which crystals of uric acid precipitate in the joints.
gout
malformations of the brain, spinal cord, or both during embryonic development.
gout
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
gout
the niacin-deficiency disease.
gout
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
gout
the vitamin C-deficiency disease.
hemorrhagic disease
a compound that protects other compounds from oxygen by itself reacting with oxygen.
hemorrhagic disease
rupture of the red blood cells, caused by vitamin E deficiency.
hemorrhagic disease
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
hemorrhagic disease
the vitamin K-deficiency disease in which blood fails to clot.
hemorrhagic disease
a bone disease characterized by softening of the bones.
hemorrhagic disease
literally, porous bones; reduced density of the bones.
hemorrhagic disease
the vitamin D-deficiency disease in children.
hemorrhagic disease
the thiamin-deficiency disease.
hemorrhagic disease
a metabolic disease in which crystals of uric acid precipitate in the joints.
hemorrhagic disease
malformations of the brain, spinal cord, or both during embryonic development.
hemorrhagic disease
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
hemorrhagic disease
the niacin-deficiency disease.
hemorrhagic disease
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
hemorrhagic disease
the vitamin C-deficiency disease.
erythrocyte hemolysis
a compound that protects other compounds from oxygen by itself reacting with oxygen.
erythrocyte hemolysis
rupture of the red blood cells, caused by vitamin E deficiency.
erythrocyte hemolysis
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
erythrocyte hemolysis
the vitamin K-deficiency disease in which blood fails to clot.
erythrocyte hemolysis
a bone disease characterized by softening of the bones.
erythrocyte hemolysis
literally, porous bones; reduced density of the bones.
erythrocyte hemolysis
the vitamin D-deficiency disease in children.
erythrocyte hemolysis
the thiamin-deficiency disease.
erythrocyte hemolysis
a metabolic disease in which crystals of uric acid precipitate in the joints.
erythrocyte hemolysis
malformations of the brain, spinal cord, or both during embryonic development.
erythrocyte hemolysis
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
erythrocyte hemolysis
the niacin-deficiency disease.
erythrocyte hemolysis
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
erythrocyte hemolysis
the vitamin C-deficiency disease.
beriberi
a compound that protects other compounds from oxygen by itself reacting with oxygen.
beriberi
rupture of the red blood cells, caused by vitamin E deficiency.
beriberi
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
beriberi
the vitamin K-deficiency disease in which blood fails to clot.
beriberi
a bone disease characterized by softening of the bones.
beriberi
literally, porous bones; reduced density of the bones.
beriberi
the vitamin D-deficiency disease in children.
beriberi
the thiamin-deficiency disease.
beriberi
a metabolic disease in which crystals of uric acid precipitate in the joints.
beriberi
malformations of the brain, spinal cord, or both during embryonic development.
beriberi
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
beriberi
the niacin-deficiency disease.
beriberi
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
beriberi
the vitamin C-deficiency disease.
Question
The best way to reap the benefits of phytochemicals is by:

A) eating a variety of plant foods.
B) taking a variety of supplements.
C) increasing your consumption of animal foods.
D) frequently consuming manufactured functional foods.
E) avoiding eggs.
Question
The first signs of vitamin C deficiency appear in the:

A) blood vessels.
B) bones.
C) hair.
D) fingernails.
E) digestive system.
Question
Phytochemicals:

A) are essential nutrients.
B) are bioactive compounds found in foods.
C) have no biological activity in the body.
D) are nonessential nutrients.
E) lose their function when cooked.
Question
Which of the following provides resveratrol?

A) soy
B) carrots
C) flaxseed
D) red wine
E) black-eyed peas
Question
What vitamin deficiency causes pernicious anemia?

A) vitamin B6
B) vitamin B12
C) folate
D) thiamin
E) riboflavin
Question
Many of the symptoms of vitamin C deficiency, such as dry scaly skin and failure of wounds to heal, are related to the role of vitamin C in the synthesis of:

A) calcium and iron.
B) thyroxin.
C) antioxidants.
D) adrenal hormones.
E) collagen.
Question
Identify factors that can place people at risk for vitamin E deficiency and explain why.
Question
The formation of collagen requires an adequate intake of:

A) vitamin K.
B) vitamin E.
C) vitamin C.
D) folate.
E) biotin.
Question
Identify three ways vitamin D helps to maintain blood concentrations of calcium and phosphorus.
Question
Which of these pairs of vitamins is the most widespread among food groups?

A) vitamin E and beta-carotene
B) pantothenic acid and biotin
C) folate and vitamin B12
D) vitamin C and vitamin D
E) thiamin and riboflavin
Question
Coenzymes are important because:

A) they function as antioxidants.
B) they facilitate vitamin absorption.
C) they function as catalysts.
D) they facilitate chemical reactions.
E) they increase the body's energy requirement.
Question
Which of the following groups of phytochemicals includes lycopene?

A) resveratrols
B) carotenoids
C) flavonoids
D) phytoestrogens
E) isothiocyanates
Question
Which of the following may result from taking megadoses of vitamin C?

A) increased excretion in urine
B) scurvy
C) gout
D) gallstones
E) weight gain
Question
The absorption of iron from iron-containing foods can double or triple when eaten at the same meal with foods containing:

A) folate.
B) protein.
C) vitamin C.
D) calcium.
E) phosphorous.
Question
Which of the following groups needs an intake of vitamin C in excess of the RDA?

A) smokers
B) professional athletes
C) the elderly
D) growing children
E) pregnant women
Question
Among the following, the best food sources for the water-soluble vitamins are:

A) butter and vegetable oils.
B) fish and cereals.
C) sugar and cornstarch.
D) egg yolks and apples.
E) soy and potatoes.
Question
Studies suggest that a diet rich in carotenoids is associated with a lower risk of:

A) diabetes.
B) arthritis.
C) heart disease.
D) osteoporosis.
E) hot flashes.
Question
Research concerning the safety and effectiveness of manufactured functional foods:

A) indicates that they should be avoided.
B) shows definite benefits to consuming such foods.
C) suggests that they may be substituted for prescription drugs.
D) is supported by the FDA.
E) is inconclusive at this time.
Question
Which of these meals is lacking in vitamin C?

A) hotdog, cabbage, French fries, and milk
B) roast beef, cheese sauce, noodles, and tea
C) roast beef, broccoli, noodles, and coffee
D) spaghetti with tomato sauce, meatballs, garlic bread, and cantaloupe
E) pork chop, baked potato with broccoli topping, and diet soda
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Deck 8: The Vitamins
1
The active form of vitamin A for vision is:

A) opsin.
B) retinoic acid.
C) retinal.
D) rhodopsin.
E) retinol-binding protein.
C
2
Which of the following is a major cause of childhood blindness worldwide?

A) vitamin C deficiency
B) riboflavin deficiency
C) pantothenic acid deficiency
D) vitamin A deficiency
E) rickets disease
D
3
Children with measles or measles-related infections may benefit from supplementation with:

A) vitamin C.
B) vitamin A.
C) iron.
D) folate.
E) vitamin E.
B
4
Vitamin A plays a role in cell differentiation. What does this mean?

A) It helps cells differentiate between the vitamins.
B) It allows cells to mature so they can perform their intended functions.
C) It promotes the storage of fat-soluble vitamins.
D) It aids in differentiating between fat- and water-soluble vitamins.
E) It causes abnormal fetal development and birth defects.
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5
Beriberi is also called the thiamin-deficiency disease.
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6
Overconsumption of the fat-soluble vitamins results in:

A) accumulation, which aids in the prevention of colds and infections.
B) no ill effects because they are excreted.
C) possible toxicity due to an accumulation in the body.
D) weight gain due to increased fat intake.
E) deficiency of water-soluble vitamins.
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7
Many of the symptoms of vitamin A deficiency, such as blindness and an increase in respiratory infections, are related to the role of vitamin A in:

A) blood clotting.
B) synthesis of visual pigments.
C) controlling osteoclast and osteoblast activity.
D) synthesis of hormones.
E) maintaining the epithelial cells.
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8
It is not absolutely necessary to consume the fat-soluble vitamins every day because they:

A) would just be excreted from the body.
B) are only absorbed in limited amounts.
C) can be stored in the body for future use.
D) would reach toxic amounts in the body.
E) cannot be absorbed every day.
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9
Vitamin A deficiency results in rickets disease in children.
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10
How are vitamins similar to carbohydrates, fats, and proteins?

A) typical roles in the body
B) organic nature
C) chemical structure
D) amounts required in the diet
E) rate of absorption
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11
Which form of vitamin A is involved in gene expression?

A) retinoic acid
B) retinol
C) retinal
D) retinol-binding protein
E) opsin
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12
In which of the following is vitamin A least likely to be involved?

A) maintaining healthy epithelial tissues
B) maintaining healthy eye tissues
C) fighting infection
D) releasing energy from the energy-yielding nutrients
E) sperm development
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13
Beta-carotene prevents which of the following from damaging cells?

A) free radicals
B) antioxidants
C) blood clots
D) vitamin E
E) calciferol
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14
Which of the following statements about fat-soluble vitamins would you be least likely to tell your client?

A) They are readily excreted from the body when consumed in excess.
B) They must be bound to proteins to travel in the blood.
C) They may be consumed less frequently than the water-soluble vitamins.
D) They may accumulate to toxic levels in the body.
E) Toxicity is more likely to occur from supplements rather than food.
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15
Excessive vitamin A during pregnancy poses a teratogenic risk. This means that:

A) it can increase the risk of birth defects.
B) it can build up in the blood and cause calcification of soft tissues.
C) it can result in a low-birth weight infant.
D) it can increase the risk of infectious disease.
E) it can result in osteomalacia.
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16
A dietary deficiency of vitamin A can produce:

A) rickets.
B) night blindness.
C) a prolonged blood-clotting time.
D) sensitivity to light.
E) birth defects.
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17
Which of the following would not be considered a fat-soluble vitamin?

A) vitamin A
B) vitamin C
C) vitamin D
D) vitamin E
E) vitamin K
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18
Bioavailability refers to the rate and extent to which a nutrient is absorbed and used.
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19
The rate and extent to which a vitamin is absorbed and used by the body is called:

A) vitamin efficiency.
B) bioavailability.
C) fortification.
D) vitamin equivalency.
E) enzymatic destruction.
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20
The fat-soluble vitamins:

A) are less stable than the water-soluble vitamins.
B) are first absorbed into the lymph.
C) are usually constituents of coenzymes.
D) are not essential.
E) are needed in frequent doses.
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21
The chemical name for vitamin E is:

A) beta-carotene.
B) cholecalciferol.
C) ascorbic acid.
D) tocopherol.
E) prothrombin.
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22
Vitamin A toxicity is likely to result from:

A) consuming too many dark green and deep orange vegetables.
B) overconsumption of whole grains.
C) consuming large amounts of fortified foods or vitamin A supplements.
D) drinking too much orange juice.
E) prescription acne treatments.
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23
Identify the food group that is the best source of beta-carotene.

A) protein foods group
B) milk group
C) vegetable group
D) grain group
E) fats and oils group
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24
Among fruits and vegetables, the best sources of beta-carotene are:

A) green or yellow, like lettuce and corn.
B) dark green or deep orange, like broccoli and sweet potatoes.
C) green, like lettuce, peas, and snap beans.
D) brightly colored, like tomatoes and lemons.
E) white, like rice and potatoes.
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25
What can be concluded from the results of research on vitamin E supplementation?

A) There is not enough evidence to recommend taking supplements to prevent heart disease.
B) Sufficient evidence exists to recommend that everyone over 40 should take a supplement.
C) Large doses are more beneficial than low doses.
D) All people who have had a heart attack should take a supplement.
E) Vitamin E can be used in place of statins in people with high cholesterol.
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26
From the list below, choose the most reliable food source of vitamin D.

A) whole grains
B) fortified breakfast cereal
C) yogurt
D) orange juice
E) raw skim milk
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27
A deficiency of which of the following vitamins can cause rickets?

A) A
B) B12
C) C
D) D
E) E
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28
The vitamin D-deficiency disease of children is:

A) xerophthalmia.
B) osteomalacia.
C) osteoporosis.
D) rickets.
E) skin cancer.
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29
Excessive intakes of vitamin D by adults may result in:

A) greater bone density.
B) mineral deposits in soft tissues such as the kidney.
C) deformity of the leg bones, ribs, and skull.
D) increased bone calcification.
E) increased risk of bone fractures.
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30
Vitamin D can be made in the body with the help of:

A) bacteria.
B) tryptophan.
C) vitamin A.
D) sunlight.
E) exercise.
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31
The effects of a deficiency of vitamin D are most readily observed in the:

A) nervous system.
B) cardiovascular system.
C) muscular system.
D) epithelial tissue.
E) skeletal system.
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32
Vitamin E:

A) functions as an effective anti-sterility agent in human beings.
B) protects vitamin A and polyunsaturated fatty acids from oxidation.
C) is needed by human beings as a dietary supplement because of the variety of functions it has in the body.
D) slows down the aging process.
E) activates blood clotting factors.
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33
Vitamin D deficiency is most likely among:

A) dark-skinned people who live in the south.
B) fair-skinned people who live in the south.
C) dark-skinned people with limited exposure to sunlight.
D) fair-skinned people with limited exposure to sunlight.
E) fair-skinned people who work outside most of the time.
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34
The main function of vitamin D in bone growth is to:

A) synthesize 7-dehydrocholesterol.
B) assist in the absorption of calcium and phosphorus.
C) mobilize calcium from the bone.
D) secrete calcitonin.
E) regulate the fat cells of adipose tissue.
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35
The major role of vitamin E in the body seems to be to:

A) aid in normal blood clotting.
B) act as an antioxidant.
C) aid in formation of normal epithelial tissue.
D) aid in protein metabolism.
E) prevent skin cancer.
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36
Which of the following factors is least likely to limit vitamin D synthesis in the body?

A) geographic location
B) season of the year
C) sunscreens
D) use of tanning beds
E) smog
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37
An excess of which of the following vitamins can cause excess calcium withdrawal from the bones?

A) A
B) B6
C) B12
D) C
E) D
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38
Osteomalacia is mainly due to a deficiency of which of the following?

A) vitamin A
B) vitamin D
C) parathyroid hormone
D) calcitonin
E) Vitamin E
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39
After 30 minutes of sun exposure, which of the following people would have synthesized more vitamin D?

A) a light-skinned person
B) a dark-skinned person
C) neither, because skin pigmentation is not related to vitamin D synthesis
D) they would both synthesize the same amount
E) a heavily-wrinkled person
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40
If your eyes have difficulty adjusting to dim light, eating which of the following foods daily might improve your vision?

A) steamed carrots
B) a peanut butter sandwich
C) brewer's yeast
D) pork chops
E) tofu
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41
Vitamin E deficiencies in human beings are associated with:

A) diseases of the pancreas.
B) low-carbohydrate weight-loss diets.
C) use of vitamin K supplements.
D) strict vegetarian (vegan) diets.
E) high-fat diets.
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42
Abuse of alcohol may lead to a severe form of thiamin deficiency called:

A) beriberi.
B) Wernicke-Korsakoff syndrome.
C) pellagra.
D) rickets.
E) atrophic gastritis.
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43
Which vitamin is synthesized in the intestine by bacteria?

A) vitamin A
B) vitamin C
C) vitamin D
D) vitamin E
E) vitamin K
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44
The most well-known role of vitamin B6 is in relation to:

A) carbohydrate and lactose metabolism.
B) fat metabolism.
C) protein and amino acid metabolism.
D) mineral metabolism.
E) cell division.
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45
Riboflavin is used by the body to:

A) facilitate energy metabolism.
B) prevent the disease pellagra.
C) provide building material for red blood cells.
D) maintain epithelial tissue.
E) synthesize niacin.
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46
Vitamin B6:

A) does not play a role in the metabolism of energy-yielding nutrients.
B) helps to regulate blood glucose.
C) is absorbed like the fat-soluble vitamins.
D) is only found in animal foods.
E) plays a major role in the formation of collagen.
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47
Thiamin is involved in:

A) formation of red blood cells.
B) blood coagulation.
C) collagen formation.
D) energy metabolism.
E) cell division.
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48
Absorption of which of the following requires the presence of intrinsic factor?

A) vitamin B6
B) vitamin D
C) vitamin B12
D) vitamin A
E) vitamin K
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49
Which vitamin can cause a pharmacological e ffect known to dilate the capillaries ?

A) thiamin
B) vitamin E
C) niacin
D) vitamin C
E) vitamin K
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50
Vitamin K has long been known for:

A) its functions in energy metabolism.
B) promoting healthy epithelial tissue.
C) its role in blood clotting.
D) enhancing calcium utilization.
E) maintaining the integrity of epithelial tissues.
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51
Which vitamin is most vulnerable to interactions with alcohol and other drugs?

A) vitamin C
B) vitamin E
C) folate
D) biotin
E) cobalamin
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52
Overconsumption of the water-soluble vitamins will likely result in:

A) a reserve supply, which will aid in the prevention of colds and infections.
B) no apparent change, because they are stored in the adipose tissues.
C) no apparent change, because they are excreted.
D) toxic accumulation in the liver.
E) a deficiency, because excess vitamins will be excreted.
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53
Which of the following statements is true of niacin?

A) It can be synthesized in the body from the amino acid tryptophan.
B) It can be used successfully to cure schizophrenia.
C) It is water soluble and therefore safe to administer in large doses.
D) It is an antioxidant.
E) A deficiency during pregnancy can lead to neural tube defects.
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54
The replacement of red blood cells and digestive tract cells depends most heavily on:

A) thiamin.
B) riboflavin.
C) niacin.
D) cobalamin.
E) folate.
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55
Which of the following groups of people is most at risk for a primary deficiency of vitamin B12?

A) the elderly
B) infants
C) athletes
D) vegans
E) pregnant women
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56
Ascorbic acid is another name for:

A) niacin.
B) thiamin.
C) vitamin C.
D) vitamin B6.
E) vitamin E.
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57
An important role of vitamin B12 is to assist folate in:

A) cell division.
B) conversion of tryptophan to niacin.
C) release of energy from food.
D) regulation of the body's use of calcium and phosphorus.
E) initiating blood clotting.
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58
Which of the following foods would make the greatest contribution to a person's intake of riboflavin?

A) oatmeal
B) milk
C) oranges
D) yellow squash
E) apples
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59
Vitamin E deficiency causes:

A) xerophthalmia.
B) rickets.
C) leukocytosis.
D) osteomalacia.
E) erythrocyte hemolysis.
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60
In which of the following foods would you find the least amount of Vitamin E?

A) margarine
B) salad dressing
C) wheat germ oil
D) butter
E) shortening
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61
Match between columns
pernicious anemia
a compound that protects other compounds from oxygen by itself reacting with oxygen.
pernicious anemia
rupture of the red blood cells, caused by vitamin E deficiency.
pernicious anemia
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
pernicious anemia
the vitamin K-deficiency disease in which blood fails to clot.
pernicious anemia
a bone disease characterized by softening of the bones.
pernicious anemia
literally, porous bones; reduced density of the bones.
pernicious anemia
the vitamin D-deficiency disease in children.
pernicious anemia
the thiamin-deficiency disease.
pernicious anemia
a metabolic disease in which crystals of uric acid precipitate in the joints.
pernicious anemia
malformations of the brain, spinal cord, or both during embryonic development.
pernicious anemia
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
pernicious anemia
the niacin-deficiency disease.
pernicious anemia
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
pernicious anemia
the vitamin C-deficiency disease.
osteoporosis
a compound that protects other compounds from oxygen by itself reacting with oxygen.
osteoporosis
rupture of the red blood cells, caused by vitamin E deficiency.
osteoporosis
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
osteoporosis
the vitamin K-deficiency disease in which blood fails to clot.
osteoporosis
a bone disease characterized by softening of the bones.
osteoporosis
literally, porous bones; reduced density of the bones.
osteoporosis
the vitamin D-deficiency disease in children.
osteoporosis
the thiamin-deficiency disease.
osteoporosis
a metabolic disease in which crystals of uric acid precipitate in the joints.
osteoporosis
malformations of the brain, spinal cord, or both during embryonic development.
osteoporosis
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
osteoporosis
the niacin-deficiency disease.
osteoporosis
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
osteoporosis
the vitamin C-deficiency disease.
pellagra
a compound that protects other compounds from oxygen by itself reacting with oxygen.
pellagra
rupture of the red blood cells, caused by vitamin E deficiency.
pellagra
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
pellagra
the vitamin K-deficiency disease in which blood fails to clot.
pellagra
a bone disease characterized by softening of the bones.
pellagra
literally, porous bones; reduced density of the bones.
pellagra
the vitamin D-deficiency disease in children.
pellagra
the thiamin-deficiency disease.
pellagra
a metabolic disease in which crystals of uric acid precipitate in the joints.
pellagra
malformations of the brain, spinal cord, or both during embryonic development.
pellagra
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
pellagra
the niacin-deficiency disease.
pellagra
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
pellagra
the vitamin C-deficiency disease.
scurvy
a compound that protects other compounds from oxygen by itself reacting with oxygen.
scurvy
rupture of the red blood cells, caused by vitamin E deficiency.
scurvy
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
scurvy
the vitamin K-deficiency disease in which blood fails to clot.
scurvy
a bone disease characterized by softening of the bones.
scurvy
literally, porous bones; reduced density of the bones.
scurvy
the vitamin D-deficiency disease in children.
scurvy
the thiamin-deficiency disease.
scurvy
a metabolic disease in which crystals of uric acid precipitate in the joints.
scurvy
malformations of the brain, spinal cord, or both during embryonic development.
scurvy
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
scurvy
the niacin-deficiency disease.
scurvy
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
scurvy
the vitamin C-deficiency disease.
niacin equivalents
a compound that protects other compounds from oxygen by itself reacting with oxygen.
niacin equivalents
rupture of the red blood cells, caused by vitamin E deficiency.
niacin equivalents
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
niacin equivalents
the vitamin K-deficiency disease in which blood fails to clot.
niacin equivalents
a bone disease characterized by softening of the bones.
niacin equivalents
literally, porous bones; reduced density of the bones.
niacin equivalents
the vitamin D-deficiency disease in children.
niacin equivalents
the thiamin-deficiency disease.
niacin equivalents
a metabolic disease in which crystals of uric acid precipitate in the joints.
niacin equivalents
malformations of the brain, spinal cord, or both during embryonic development.
niacin equivalents
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
niacin equivalents
the niacin-deficiency disease.
niacin equivalents
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
niacin equivalents
the vitamin C-deficiency disease.
osteomalacia
a compound that protects other compounds from oxygen by itself reacting with oxygen.
osteomalacia
rupture of the red blood cells, caused by vitamin E deficiency.
osteomalacia
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
osteomalacia
the vitamin K-deficiency disease in which blood fails to clot.
osteomalacia
a bone disease characterized by softening of the bones.
osteomalacia
literally, porous bones; reduced density of the bones.
osteomalacia
the vitamin D-deficiency disease in children.
osteomalacia
the thiamin-deficiency disease.
osteomalacia
a metabolic disease in which crystals of uric acid precipitate in the joints.
osteomalacia
malformations of the brain, spinal cord, or both during embryonic development.
osteomalacia
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
osteomalacia
the niacin-deficiency disease.
osteomalacia
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
osteomalacia
the vitamin C-deficiency disease.
antioxidant
a compound that protects other compounds from oxygen by itself reacting with oxygen.
antioxidant
rupture of the red blood cells, caused by vitamin E deficiency.
antioxidant
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
antioxidant
the vitamin K-deficiency disease in which blood fails to clot.
antioxidant
a bone disease characterized by softening of the bones.
antioxidant
literally, porous bones; reduced density of the bones.
antioxidant
the vitamin D-deficiency disease in children.
antioxidant
the thiamin-deficiency disease.
antioxidant
a metabolic disease in which crystals of uric acid precipitate in the joints.
antioxidant
malformations of the brain, spinal cord, or both during embryonic development.
antioxidant
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
antioxidant
the niacin-deficiency disease.
antioxidant
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
antioxidant
the vitamin C-deficiency disease.
neural tube defects
a compound that protects other compounds from oxygen by itself reacting with oxygen.
neural tube defects
rupture of the red blood cells, caused by vitamin E deficiency.
neural tube defects
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
neural tube defects
the vitamin K-deficiency disease in which blood fails to clot.
neural tube defects
a bone disease characterized by softening of the bones.
neural tube defects
literally, porous bones; reduced density of the bones.
neural tube defects
the vitamin D-deficiency disease in children.
neural tube defects
the thiamin-deficiency disease.
neural tube defects
a metabolic disease in which crystals of uric acid precipitate in the joints.
neural tube defects
malformations of the brain, spinal cord, or both during embryonic development.
neural tube defects
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
neural tube defects
the niacin-deficiency disease.
neural tube defects
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
neural tube defects
the vitamin C-deficiency disease.
free radicals
a compound that protects other compounds from oxygen by itself reacting with oxygen.
free radicals
rupture of the red blood cells, caused by vitamin E deficiency.
free radicals
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
free radicals
the vitamin K-deficiency disease in which blood fails to clot.
free radicals
a bone disease characterized by softening of the bones.
free radicals
literally, porous bones; reduced density of the bones.
free radicals
the vitamin D-deficiency disease in children.
free radicals
the thiamin-deficiency disease.
free radicals
a metabolic disease in which crystals of uric acid precipitate in the joints.
free radicals
malformations of the brain, spinal cord, or both during embryonic development.
free radicals
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
free radicals
the niacin-deficiency disease.
free radicals
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
free radicals
the vitamin C-deficiency disease.
rickets
a compound that protects other compounds from oxygen by itself reacting with oxygen.
rickets
rupture of the red blood cells, caused by vitamin E deficiency.
rickets
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
rickets
the vitamin K-deficiency disease in which blood fails to clot.
rickets
a bone disease characterized by softening of the bones.
rickets
literally, porous bones; reduced density of the bones.
rickets
the vitamin D-deficiency disease in children.
rickets
the thiamin-deficiency disease.
rickets
a metabolic disease in which crystals of uric acid precipitate in the joints.
rickets
malformations of the brain, spinal cord, or both during embryonic development.
rickets
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
rickets
the niacin-deficiency disease.
rickets
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
rickets
the vitamin C-deficiency disease.
gout
a compound that protects other compounds from oxygen by itself reacting with oxygen.
gout
rupture of the red blood cells, caused by vitamin E deficiency.
gout
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
gout
the vitamin K-deficiency disease in which blood fails to clot.
gout
a bone disease characterized by softening of the bones.
gout
literally, porous bones; reduced density of the bones.
gout
the vitamin D-deficiency disease in children.
gout
the thiamin-deficiency disease.
gout
a metabolic disease in which crystals of uric acid precipitate in the joints.
gout
malformations of the brain, spinal cord, or both during embryonic development.
gout
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
gout
the niacin-deficiency disease.
gout
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
gout
the vitamin C-deficiency disease.
hemorrhagic disease
a compound that protects other compounds from oxygen by itself reacting with oxygen.
hemorrhagic disease
rupture of the red blood cells, caused by vitamin E deficiency.
hemorrhagic disease
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
hemorrhagic disease
the vitamin K-deficiency disease in which blood fails to clot.
hemorrhagic disease
a bone disease characterized by softening of the bones.
hemorrhagic disease
literally, porous bones; reduced density of the bones.
hemorrhagic disease
the vitamin D-deficiency disease in children.
hemorrhagic disease
the thiamin-deficiency disease.
hemorrhagic disease
a metabolic disease in which crystals of uric acid precipitate in the joints.
hemorrhagic disease
malformations of the brain, spinal cord, or both during embryonic development.
hemorrhagic disease
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
hemorrhagic disease
the niacin-deficiency disease.
hemorrhagic disease
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
hemorrhagic disease
the vitamin C-deficiency disease.
erythrocyte hemolysis
a compound that protects other compounds from oxygen by itself reacting with oxygen.
erythrocyte hemolysis
rupture of the red blood cells, caused by vitamin E deficiency.
erythrocyte hemolysis
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
erythrocyte hemolysis
the vitamin K-deficiency disease in which blood fails to clot.
erythrocyte hemolysis
a bone disease characterized by softening of the bones.
erythrocyte hemolysis
literally, porous bones; reduced density of the bones.
erythrocyte hemolysis
the vitamin D-deficiency disease in children.
erythrocyte hemolysis
the thiamin-deficiency disease.
erythrocyte hemolysis
a metabolic disease in which crystals of uric acid precipitate in the joints.
erythrocyte hemolysis
malformations of the brain, spinal cord, or both during embryonic development.
erythrocyte hemolysis
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
erythrocyte hemolysis
the niacin-deficiency disease.
erythrocyte hemolysis
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
erythrocyte hemolysis
the vitamin C-deficiency disease.
beriberi
a compound that protects other compounds from oxygen by itself reacting with oxygen.
beriberi
rupture of the red blood cells, caused by vitamin E deficiency.
beriberi
highly reactive chemical forms that can cause destructive changes in nearby compounds, sometimes setting up a chain reaction.
beriberi
the vitamin K-deficiency disease in which blood fails to clot.
beriberi
a bone disease characterized by softening of the bones.
beriberi
literally, porous bones; reduced density of the bones.
beriberi
the vitamin D-deficiency disease in children.
beriberi
the thiamin-deficiency disease.
beriberi
a metabolic disease in which crystals of uric acid precipitate in the joints.
beriberi
malformations of the brain, spinal cord, or both during embryonic development.
beriberi
the amount of niacin present in food, including the niacin that can theoretically be made from tryptophan, its precursor, present in the food.
beriberi
the niacin-deficiency disease.
beriberi
a vitamin B12-deficiency disease caused by lack of intrinsic factor and characterized by large, immature red blood cells and damage to the nervous system.
beriberi
the vitamin C-deficiency disease.
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62
The best way to reap the benefits of phytochemicals is by:

A) eating a variety of plant foods.
B) taking a variety of supplements.
C) increasing your consumption of animal foods.
D) frequently consuming manufactured functional foods.
E) avoiding eggs.
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63
The first signs of vitamin C deficiency appear in the:

A) blood vessels.
B) bones.
C) hair.
D) fingernails.
E) digestive system.
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64
Phytochemicals:

A) are essential nutrients.
B) are bioactive compounds found in foods.
C) have no biological activity in the body.
D) are nonessential nutrients.
E) lose their function when cooked.
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65
Which of the following provides resveratrol?

A) soy
B) carrots
C) flaxseed
D) red wine
E) black-eyed peas
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66
What vitamin deficiency causes pernicious anemia?

A) vitamin B6
B) vitamin B12
C) folate
D) thiamin
E) riboflavin
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67
Many of the symptoms of vitamin C deficiency, such as dry scaly skin and failure of wounds to heal, are related to the role of vitamin C in the synthesis of:

A) calcium and iron.
B) thyroxin.
C) antioxidants.
D) adrenal hormones.
E) collagen.
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68
Identify factors that can place people at risk for vitamin E deficiency and explain why.
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69
The formation of collagen requires an adequate intake of:

A) vitamin K.
B) vitamin E.
C) vitamin C.
D) folate.
E) biotin.
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70
Identify three ways vitamin D helps to maintain blood concentrations of calcium and phosphorus.
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71
Which of these pairs of vitamins is the most widespread among food groups?

A) vitamin E and beta-carotene
B) pantothenic acid and biotin
C) folate and vitamin B12
D) vitamin C and vitamin D
E) thiamin and riboflavin
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72
Coenzymes are important because:

A) they function as antioxidants.
B) they facilitate vitamin absorption.
C) they function as catalysts.
D) they facilitate chemical reactions.
E) they increase the body's energy requirement.
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73
Which of the following groups of phytochemicals includes lycopene?

A) resveratrols
B) carotenoids
C) flavonoids
D) phytoestrogens
E) isothiocyanates
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74
Which of the following may result from taking megadoses of vitamin C?

A) increased excretion in urine
B) scurvy
C) gout
D) gallstones
E) weight gain
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75
The absorption of iron from iron-containing foods can double or triple when eaten at the same meal with foods containing:

A) folate.
B) protein.
C) vitamin C.
D) calcium.
E) phosphorous.
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76
Which of the following groups needs an intake of vitamin C in excess of the RDA?

A) smokers
B) professional athletes
C) the elderly
D) growing children
E) pregnant women
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77
Among the following, the best food sources for the water-soluble vitamins are:

A) butter and vegetable oils.
B) fish and cereals.
C) sugar and cornstarch.
D) egg yolks and apples.
E) soy and potatoes.
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78
Studies suggest that a diet rich in carotenoids is associated with a lower risk of:

A) diabetes.
B) arthritis.
C) heart disease.
D) osteoporosis.
E) hot flashes.
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79
Research concerning the safety and effectiveness of manufactured functional foods:

A) indicates that they should be avoided.
B) shows definite benefits to consuming such foods.
C) suggests that they may be substituted for prescription drugs.
D) is supported by the FDA.
E) is inconclusive at this time.
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80
Which of these meals is lacking in vitamin C?

A) hotdog, cabbage, French fries, and milk
B) roast beef, cheese sauce, noodles, and tea
C) roast beef, broccoli, noodles, and coffee
D) spaghetti with tomato sauce, meatballs, garlic bread, and cantaloupe
E) pork chop, baked potato with broccoli topping, and diet soda
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