Exam 13: Failures of Host Defense Mechanisms
Exam 1: Basic Concepts in Immunology44 Questions
Exam 2: Innate Immunity: the First Lines of Defense32 Questions
Exam 3: The Induced Responses of Innate Immunity39 Questions
Exam 4: Antigen Recognition by B-Cell and T-Cell Receptors28 Questions
Exam 5: The Generation of Lymphocyte Antigen Receptors33 Questions
Exam 6: Antigen Presentation to T Lymphocytes30 Questions
Exam 7: Lymphocyte Receptor Signaling42 Questions
Exam 8: Development and Survival of Lymphocytes37 Questions
Exam 9: T-Cell-Mediated Immunity37 Questions
Exam 10: The Humoral Immune Response30 Questions
Exam 11: Integrated Dynamics of Innate and Adaptive Immunity28 Questions
Exam 12: The Mucosal Immune System27 Questions
Exam 13: Failures of Host Defense Mechanisms43 Questions
Exam 14: Allergy and Allergic Diseases26 Questions
Exam 15: Autoimmunity and Transplantation31 Questions
Exam 16: Manipulation of the Immune Response34 Questions
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Human genome-wide genetic association (GWAS) studies have identified several genetic polymorphisms that contribute to HIV control, and the rate of disease progression to AIDS. Which two immune cell subsets are implicated by these data as important regulators of HIV replication and the rate of disease progression?
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Defects in components of the complement pathway do not lead to recurrent or persistent virus infections.
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A common misconception is that our immune system fails to make a productive immune response to HIV infection, thereby leading to chronic infection. In fact, following a primary HIV infection, our immune system:
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