Exam 11: Memory Consolidation

Which statement(s) is/are false? (Select all that apply.)

A drug inhibits molecule X, and it impaired performance at the long, 24-hour retention interval but had no effect on performance at the short, 1-hour retention interval. What can be said about the contribution of molecule X to memory?

What are the differences between mTORC1 and mTORC2?

That rapamycin administers just before training interfered with retention two and seven days later indicates that the mTOR pathway is critically for at least two ways of protein synthesis.

The TrkB receptor is activated by the first messenger glutamate.

What is the retention interval?

C/EBPβ is critical to the first wave of protein synthesis.

Describe two sources of evidence that BDNF is important for memory consolidation.

Rapamycin administer prior to training interfered with retention two and seven days later.

Why is the rictor KO mouse useful?

What is the evidence that the UPS is involved in memory consolidation?

C/EBPβ is targeted by CaMKII for transcription.

To determine the role of a molecule in memory the experiment should use at least two retention intervals.

Inhibiting the mTORC1 complex _______. (Select all that apply.)

The decay rate for long-term memories is more rapid than the decay rate for short-term memories.

What is the role of C/EBPβ in the autoregulatory loop?

Which statement(s) is/are true? (Select all that apply.)