Exam 46: Shock, Multiple Organ Dysfunction Syndrome, and Burns in Adult

In septic shock, which mediators are anti-inflammatory?

For which type of shock would antihistamines and corticosteroids be prescribed?

Match the types of shock with the corresponding descriptions. Terms can be used more than once. -Follows infection

What mechanism causes organ injury in primary MODS?

Daily evaporative water loss following burn injury is approximately _____ times normal.

In primary multiple organ dysfunction syndrome (MODS), organ injury is directly associated with impaired perfusion.

What is the clinical hallmark of neurogenic shock due to overstimulation of the parasympathetic nervous system?

The level of IL-1 detected in the serum of a burned individual correlates directly with the burn survival; high levels are associated with a higher mortality.

The clinical manifestations of failure of individual organs in MODS are caused only by inflammatory mediator damage, tissue hypoxia, and hypermetabolism.

The final common pathway in all shock is:

What type of shock develops with overstimulation of the parasympathetic nervous system or understimulation of the sympathetic nervous system?

To compensate for hypovolemic shock, the liver and spleen add to the blood volume by disgorging stored red blood cells and plasma.

Blistering of the skin occurs in _____ burns.

When proteins are broken down anaerobically, ammonia and urea are produced.

The cause of organ failure in shock is depletion of protein.

In secondary MODS, what stimulates the normal endothelial cells to change to a proinflammatory state?

With reperfusion in MODS, oxygen radicals are formed from oxygen by the action of xanthine oxidase that attack the already damaged tissue.

The fluid most often used in fluid resuscitation following a major burn injury is:

Acute burn injury results in _____ shock.

The endpoint of burn shock is defined as the time when the individual is able to: